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oai:open-archive.highwire.org:amjepid:146/4/2872015-05-11HighWireOUPamjepid:146:4
Anthropometric Measurements and Vertebral Deformities
Johnell, O.
O'Neill, T.
Felsenberg, D.
Kanis, J.
Cooper, C.
Silman, A. J.
the European Vertebral Osteoporosis Study (EVOS) Group,
ORIGINAL CONTRIBUTIONS
To investigate the association between anthropometric indices and morphometrically determined vertebral deformity, the authors carried out a cross-sectional study using data from the European Vertebral Osteoporosis Study (EVOS), a population-based study of vertebral osteoporosis in 36 European centers from 19 countries. A total of 16,047 EVOS subjects were included in this analysis, of whom 1,973 subjects (915 males, 1,058 females) (12.3%) aged 50 years or over had one or more vertebral deformities (“cases”). The cases were compared with the 14,074 subjects (6,539 males, 7,535 females) with morphometrically normal spines (“controls”). Data were collected on self-reported height at age 25 years and minimum weight after age 25 years, as well as on current measured height and weight. Body mass index (BMI) and height and weight change were calculated from these data. The relations between these variables and vertebral deformity were examined separately by sex with logistic regression adjusting for age, smoking, and physical activity. In females, there was a significant trend of decreasing risk with increasing quintile of current weight, current BMI, and weight gain since age 25 years. In males, subjects in the lightest quintile for these measures were at increased risk but there was no evidence of a trend. An ecologic analysis by country revealed a negative correlation between mean BMI and the prevalence of deformity in females but not in males. The authors conclude that low body weight is associated with presence of vertebral deformity. <it>Am J Epidemiol</it> 1997;146:287–93.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/287
http://dx.doi.org/10.1093/oxfordjournals.aje.a009269
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/2942015-05-11HighWireOUPamjepid:146:4
Association of Soy and Fiber Consumption with the Risk of Endometrial Cancer
Goodman, Marc T.
Wilkens, Lynne R.
Hankin, Jean H.
Lyu, Li-Ching
Wu, Anna H.
Kolonel, Laurence N.
ORIGINAL CONTRIBUTIONS
The authors conducted a case-control study among the multi-ethnic population of Hawaii to examine the role of dietary soy, fiber, and related foods and nutrients on the risk of endometrial cancer. Endometrial cancer cases (<it>n</it> = 332) diagnosed between 1985 and 1993 were identified from the five main ethnic groups in the state (Japanese, Caucasian, Native Hawaiian, Filipino, and Chinese) through the rapid-reporting system of the Hawaii Tumor Registry. Population controls (<it>n</it> = 511) were selected randomly from lists of female Oahu residents and matched to cases on age (±2.5 years) and ethnicity. All subjects were interviewed using a diet history questionnaire that included over 250 food items. Non-dietary risk factors for endometrial cancer included nulliparity, never using oral contraceptives, fertility drug use, use of unopposed estrogens, a history of diabetes mellitus or hypertension, and a high Quetelet's index (kg/m2). Energy intake from fat, but not from other sources, was positively associated with the risk of endometrial cancer. The authors also found a positive, monotonic relation of fat intake with the odds ratios for endometrial cancer after adjustment for energy intake. The consumption of fiber, but not starch, was inversely related to risk after adjustment for energy intake and other confounders. Similar inverse gradients in the odds ratios were obtained for crude fiber, non-starch polysaccharide, and dietary fiber. Sources of fiber, including cereal and vegetable and fruit fiber, were associated with a 29–46% reduction in risk for women in the highest quartiles of consumption. Vitamin A and possibly vitamin C, but not vitamin E, were also inversely associated with endometrial cancer, although trends were not strong. High consumption of soy products and other legumes was associated with a decreased risk of endometrial cancer (<it>p</it> for trend = 0.01; odds ratio = 0.46, 95% confidence interval 0.26–0.83) for the highest compared with the lowest quartile of soy intake. Similar reductions in risk were found for increased consumption of other sources of phytoestrogens such as whole grains, vegetables, fruits, and seaweeds. Ethnic-specific analyses were generally consistent with these results. The observed dietary associations appeared to be largely independent of other risk factors, although the effects of soy and legumes on risk were limited to women who were never pregnant or who had never used unopposed estrogens. These data suggest that plant-based diets low in calories from fat, high in fiber, and rich in legumes (especially soybeans), whole grain foods, vegetables, and fruits reduce the risk of endometrial cancer. These dietary associations may explain in part the reduced rates of uterine cancer in Asian countries compared with those in the United States. <it>Am J Epidemiol</it> 1997;146:294–306.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/294
http://dx.doi.org/10.1093/oxfordjournals.aje.a009270
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3072015-05-11HighWireOUPamjepid:146:4
Breast Cancer Risk Factors According to Combined Estrogen and Progesterone Receptor Status: A Case-Control Analysis
Yoo, Keun-Young
Tajima, Kazuo
Miura, Shigeto
Takeuchi, Tohru
Hirose, Kaoru
Risch, Harvey
Dubrow, Robert
ORIGINAL CONTRIBUTIONS
Breast cancers demonstrate a gradient of responsiveness to endocrine therapy according to hormone receptor status, with tumors positive for both estrogen and progesterone receptors responding most favorably. The authors hypothesized that reproductive risk factors, which are probably mediated by endogenous hormones, would also differ according to receptor status, and that tumors positive for both receptors would exhibit the highest risk. Information on risk factors was obtained from 1,154 breast cancer cases and 21,714 cancer-free controls at the Aichi Cancer Center Hospital, Nagoya, Japan between 1988 and 1992. Receptor status was known for 40% of cases. For a given risk factor, odds ratios with respect to the common control group for breast cancers of differing receptor status were compared using multiple polytomous logistic regression. Risk factors did not differ significantly according to estrogen receptor status. However, age at diagnosis/interview, occupation, age at menarche, menstrual regularity at ages 20–29 years, and cigarette smoking differed significantly or borderline significantly in effect according to progesterone receptor status. Stratification of cases according to joint estrogen and progesterone receptor status indicated that estrogen receptor status did not modify this difference in effect. This study did not support the hypothesis of a gradient of risk for reproductive factors according to hormone receptor status. The authors recommend that the finding that some risk factors differ according to progesterone receptor status should be pursued in further studies. <it>Am J Epidemiol</it> 1997;146:307-14.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/307
http://dx.doi.org/10.1093/oxfordjournals.aje.a009271
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3152015-05-11HighWireOUPamjepid:146:4
Low Insulin Sensitivity is Associated with Clustering of Cardiovascular Disease Risk Factors
Mykkänen, Leena
Haffner, Steven M.
Rönnemaa, Tapani
Bergman, Richard N.
Laakso, Markku
ORIGINAL CONTRIBUTIONS
Hyperinsulinemia is associated with multiple metabolic disorders including high triglyceride level, low high density lipoprotein (HDL) cholesterol level, hypertension, and impaired glucose tolerance (IGT). This metabolic constellation is also called the insulin resistance syndrome. All previous data on clustering of these risk factors are, however, based on insulin levels. Therefore, the authors examined the association of insulin sensitivity estimated by means of a frequently sampled intravenous glucose tolerance test and the minimal model with the number of metabolic disorders (dyslipidemia [high triglyceride level or low HDL cholesterol level or both], hypertension, and IGT according to the World Health Organization criteria). Of 153 nondiabetic subjects aged 53–61 years who had participated in a previous population-based study, 79 had no disorders, 55 had one disorder, 16 had two disorders, and 3 had three disorders. Insulin sensitivity index (S<inf>1</inf>) decreased with the increasing number of disorders (4.1, 3.5, 1.8, and 1.4×10−4 min−1 μU−1 mL−1, in subjects with 0, 1, 2, and 3 disorders, respectively; <it>p</it><0.001 for trend). Similarly, fasting (7.5, 7.8, 15.3, and 22.0 μU/mL; <it>p</it><0.001) and 2-hour insulin levels (39.9, 49.0, 98.7, and 149.6 μU/mL; <it>p</it><0.001) increased by the increasing number of disorders. The relations of S<inf>1</inf> and fasting and 2-hour insulin levels with multiple metabolic disorders were independent of sex, obesity, and body fat distribution. Furthermore, these associations were similar in men and women and in lean and obese subjects. The authors conclude that a clustering of cardiovascular disease risk factors in nondiabetic subjects is not only associated with hyperinsulinemia but also with insulin resistance. <it>Am J Epidemiol</it> 1997;146:315-21.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/315
http://dx.doi.org/10.1093/oxfordjournals.aje.a009272
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3222015-05-11HighWireOUPamjepid:146:4
Physical Activity and Cardiovascular Disease Risk Profile in Women
Pols, Margreet A.
Peeters, Petra H. M.
Twisk, Jos W. R.
Kemper, Han C. G.
Grobbee, Diederick E.
ORIGINAL CONTRIBUTIONS
In a population of 4,576 Dutch women aged 49–70 years who participated in the European Prospective Investigation into Cancer and Nutrition (EPIC) in 1993–1995, the relation between physical activity and the presence of cardiovascular disease risk indicators was assessed cross-sectionally. Physical activity was determined from a self-administered questionnaire, while blood pressure, heart rate, body mass index, waist/hip ratio, and waist circumference were measured at the study center. Mean risk indicator levels were calculated for different activity categories. Blood pressure was most clearly associated with time spent in sports (mean systolic blood pressure, adjusted for age, level of education, and smoking, 128.9 mmHg in the highest sports tertile, and 132.1 mmHg in the lowest sports tertile; mean diastolic blood pressure, 77.8 mmHg and 79.0 mmHg, respectively). Body mass index, waist/hip ratio, and waist circumference showed an inverse relation with cycling, gardening, do-it-yourself-activities, and sports. In this population, leisure-time activity was inversely related to cardiovascular disease risk indicators, but work activity and housework were not. The authors conclude that if investigators wish to measure physical activity in women over age 50 years with the aim to identify high- and low-risk groups for cardiovascular disease, they should consider not only housework activity, but also leisure-time activities such as cycling, sports, and do-it-yourself activities. <it>Am J Epidemiol</it> 1997;146:322-8.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/322
http://dx.doi.org/10.1093/oxfordjournals.aje.a009273
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3292015-05-11HighWireOUPamjepid:146:4
Gestational Age, Birth Weight, and Perinatal Death among Births to Norwegian Farmers, 1967-1991
Kristensen, Petter
Irgens, Lorentz M.
Anderson, Aage
Bye, Anne Snellingen
Sundheim, Leif
ORIGINAL CONTRIBUTIONS
Perinatal health was investigated by linkage with the Medical Birth Registry of Norway for 192,417 births that took place between 1967 and 1991 among parents identified as farm holders in Norwegian agricultural censuses in 1969–1989. In a comparison with 61,351 births to nonfarmers in agricultural municipalities, farmers' births had an advantageous distribution of gestational ages and birth weights. Perinatal mortality was similar in the two groups, but the proportion of late-term abortions (gestational weeks 16–27) was higher among farmers' births (odds ratio (OR) = 1.9, 95% confidence interval (CI) 1.6–2.3). Exposure indicators were classified on the basis of information given in the agricultural censuses and climate data for the grain harvest seasons of 1966–1991. The main hypotheses were that perinatal death is associated with parental exposure to pesticides, <it>Toxoplasma</it> contracted from infected sheep or pigs, or mycotoxins found in grain farming. There was no convincing evidence that perinatal death is associated with use of pesticides, sheep farming, or pig farming. The increase in late-term abortion among the farmers could to some extent be attributed to an excess of midpregnancy (weeks 21–24) deliveries among grain farmers; grain farmers had 132 deliveries at this time in pregnancy (2.8 per 1,000 pregnancies), while the nongrain farmers had 236 deliveries in midpregnancy (1.8 per 1,000). The authors found odds ratios (95% Cl) that indicated that grain farming risk was higher after the harvest (1.8, 1.1–2.8), in seasons with a poor quality harvest (2.4, 1.5–3.8), and in pregnancies with multiple births (3.8, 1.7–8.2). These results support the hypothesis that occupational exposure to mycotoxins in grain induces labor at an early stage of pregnancy. <it>Am J Epidemiol</it> 1997;146:329-38.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/329
http://dx.doi.org/10.1093/oxfordjournals.aje.a009274
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3392015-05-11HighWireOUPamjepid:146:4
Hypothesis Concerning the U-shaped Relation between Body Mass Index and Mortality
Allison, David B.
Faith, Myles S.
Heo, Moonseong
Kotler, Donald P.
ORIGINAL CONTRIBUTIONS
Numerous studies have documented a U- or J-shaped association between body mass index (BMI) (kg/m2) and mortality, such that increased mortality rate is associated with relatively low and high BMI values. It has been argued elsewhere that the elevated mortality rate observed at lower BMI values actually results from the effects of unmeasured confounding variables, in particular smoking status and preexisting disease. In this paper, the authors present an additional explanation for the phenomenon, i.e., nonspecific measurement. They propose that differential health consequences of fat mass and fat-free mass can be masked by the use of BMI when studied in relation to mortality. To illustrate this point, they use body composition data from 1,137 healthy adults and specify a hypothetical underlying BMI-mortality model in which the logit of death increased linearly with fat mass and decreased linearly with fat-free mass, and % fat increased monotonically with BMI. The results indicate that, even under these specifications, the authors can recover a U-shaped association between BMI and mortality. Consistent with previous suggestions in the literature, future epidemiologic studies that examine the association between adiposity and mortality should prioritize the use of body composition measures. <it>Am J Epidemiol</it> 1997;146:339-49.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/339
http://dx.doi.org/10.1093/oxfordjournals.aje.a009275
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3502015-05-11HighWireOUPamjepid:146:4
Heterosexual Transmission of Human Immunodeficiency Virus (HIV) in Northern California: Results from a Ten-year Study
Padian, Nancy S.
Shiboski, Stephen C.
Glass, Sarah O.
Vittinghoff, Eric
ORIGINAL CONTRIBUTIONS
To examine rates of and risk factors for heterosexual transmission of human immunodeficiency virus (HIV), the authors conducted a prospective study of infected individuals and their heterosexual partners who have been recruited since 1985. Participants were recruited from health care providers, research studies, and health departments throughout Northern California, and they were interviewed and examined at various study clinic sites. A total of 82 infected women and their male partners and 360 infected men and their female partners were enrolled. Over 90% of the couples were monogamous for the year prior to entry into the study; <3% had a current sexually transmitted disease (STD). The median age of participants was 34 years, and the majority were white. Over 3,000 couple-months of data were available for the follow-up study. Overall, 68 (19%) of the 360 female partners of HIV-infected men (95% confidence interval (CI) 15.0–23.3%) and two (2.4%) of the 82 male partners of HIV-infected women (95% CI 0.3–8.57%) were infected. History of sexually transmitted diseases was most strongly associated with transmission. Male-to-female transmission was approximately eight times more efficient than female-to-male transmission and male-to-female per contact infectivity was estimated to be 0.0009 (95% CI 0.0005–0.001). Overtime, the authors observed increased condom use (<it>p</it> < 0.001) and no new infections. Infectivity for HIV through heterosexual transmission is low, and STDs may be the most important cofactor for transmission. Significant behavior change over time in serodiscordant couples was observed. <it>Am J Epidemiol</it> 1997;146:350-7.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/350
http://dx.doi.org/10.1093/oxfordjournals.aje.a009276
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3572015-05-11HighWireOUPamjepid:146:4
RE: "EPIDEMIOLOGY OF INSULIN-LIKE GROWTH FACTOR-I IN ELDERLY MEN AND WOMEN: THE RANCHO BERNARDO STUDY"
ERRATUM
The <it>Journal</it> has been informed by Drs. Goodman-Gruen and Barrett-Connor of errors in the abstract of their recently published paper on insulin-like growth factor-I (IGF-I) in elderly men and women (1). Specifically, as published, a sentence in the abstract reads:“IGF-I decreased linearly with age in both sexes, with significantly lower levels in men than women (126.9 μg/liter vs. 134.1 μg/liter <it>p</it> = 0.03).” The correct sentence should read: “IGF-I decreased linearly with age in both sexes, with significantly higher levels in men than women (134.1 μg/liter vs. 126.9 μg/liter; <it>p</it> = 0.03).” The <it>Journal</it> regrets these errors.
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/357
http://dx.doi.org/10.1093/oxfordjournals.aje.a009277
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3582015-05-11HighWireOUPamjepid:146:4
RE: "CRYPTOSPORIDIOSIS AMONG PATIENTS INFECTED WITH HUMAN IMMUNODEFICIENCY VIRUS: FACTORS RELATED TO SYMPTOMATIC INFECTION AND SURVIVAL"
Clark, Rebecca A.
Bessinger, Ruth
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/358
http://dx.doi.org/10.1093/oxfordjournals.aje.a009278
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3592015-05-11HighWireOUPamjepid:146:4
RE: "WATER, WASTE, AND WELL-BEING: A MULTICOUNTRY STUDY"
Cairncross, Sandy
Kolsky, Peter J.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
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http://aje.oxfordjournals.org/cgi/content/short/146/4/359
http://dx.doi.org/10.1093/oxfordjournals.aje.a009279
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3602015-05-11HighWireOUPamjepid:146:4
THE AUTHOR REPLIES
Esrey, Steven A.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/360
http://dx.doi.org/10.1093/oxfordjournals.aje.a009280
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3612015-05-11HighWireOUPamjepid:146:4
RE: "EXPOSURE TO POLYCHLORINATED DIOXINS AND FURANS (PCDD/F) AND MORTALITY IN A COHORT OF WORKERS FROM A HERBICIDE-PRODUCING PLANT IN HAMBURG, FEDERAL REPUBLIC OF GERMANY"
Swaen, Gerard M. H.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/361
http://dx.doi.org/10.1093/oxfordjournals.aje.a009281
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3622015-05-11HighWireOUPamjepid:146:4
THE FIRST AUTHOR REPLIES
Flesch-Janys, Dieter
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/362
http://dx.doi.org/10.1093/oxfordjournals.aje.a009282
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3632015-05-11HighWireOUPamjepid:146:4
RE: "A NEW PERSPECTIVE ON JOHN SNOW'S COMMUNICABLE DISEASE THEORY"
Vandenbroucke, Jan P.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/363
http://dx.doi.org/10.1093/oxfordjournals.aje.a009283
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3642015-05-11HighWireOUPamjepid:146:4
RE: "FAMILY HISTORY OF CANCER AND RISK OF LUNG CANCER AMONG LIFETIME NONSMOKING WOMEN IN THE UNITED STATES"
Foulkes, William D.
Narod, Steven A.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/364
http://dx.doi.org/10.1093/oxfordjournals.aje.a009284
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/365-a2015-05-11HighWireOUPamjepid:146:4
RE: "SEX DIFFERENCE IN HIGH DENSITY LIPOPROTEIN CHOLESTEROL IN SIX COUNTRIES"
Kesteloot, H.
Sasaki, S.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/365-a
http://dx.doi.org/10.1093/oxfordjournals.aje.a009286
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3652015-05-11HighWireOUPamjepid:146:4
RE: "MORTON LEVIN (1904-1995): HISTORY IN THE MAKING"
Terris, Milton
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/365
http://dx.doi.org/10.1093/oxfordjournals.aje.a009285
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3662015-05-11HighWireOUPamjepid:146:4
RE: ELECTROMAGNETIC FIELD EXPOSURE AND LUNG CANCER
Henshaw, Denis L.
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/366
http://dx.doi.org/10.1093/oxfordjournals.aje.a009287
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3672015-05-11HighWireOUPamjepid:146:4
BOOK REVIEWS
Greenland, Sander
BOOK REVIEWS
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/367
http://dx.doi.org/10.1093/oxfordjournals.aje.a009288
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/3702015-05-11HighWireOUPamjepid:146:4
BOOK REVIEWS
Vollmer, William M.
BOOK REVIEWS
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/370
http://dx.doi.org/10.1093/oxfordjournals.aje.a009289
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/365a1997-08-15HighWireOUPamjepid:146:4
Re: "Sex difference in high density lipoprotein cholesterol in six countries"
Kesteloot, H
Sasaki, S
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/365a
en
Copyright (C) 1997, Oxford University Press
oai:open-archive.highwire.org:amjepid:146/4/365b1997-08-15HighWireOUPamjepid:146:4
Re:"Morton Levin (1904-1995): history in the making"
Terris, M
LETTERS TO THE EDITOR
Oxford University Press
1997-08-15 00:00:00.0
TEXT
text/html
http://aje.oxfordjournals.org/cgi/content/short/146/4/365b
en
Copyright (C) 1997, Oxford University Press